Free radicals are considered the fundamental mediators of doxorubicin-induced cardiotoxicity. The semiquinone form, produced by the reduction of doxorubicin, interacts with molecular oxygen yielding ...
Insulin-like growth factor-1 (IGF-1) inhibited N-acetylsphingosine (C2-ceramide)-induced HL-60 cell apoptosis via relieving oxidative damage. This inhibitory action of IGF-1 was blocked by a ...
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